Scientific Summary
Early in 2003 Sinofresh Healthcare, Inc. conducted a series of in-vitro kill studies at Nelson Laboratories in Salt Lake City, Utah. These studies were to evaluate the product’s efficacy in killing a wide variety of yeasts, molds and bacteria. The results were encouraging and suggested that the next studies should target more specific pathogenic organisms.

In 2005, Sinofresh Healthcare, Inc. decided to take a closer look at Methicillin Resistant Staphylococcus Aureus (MRSA). These studies were in-vitro studies conducted at the Public Health Research Institute in New Jersey. In an effort to more closely mimic the nasal mucosa, the studies were conducted in presence and absence of mucin. The results were very similar in that the product exhibited its ability to kill the MRSA organisms with only a slight time delay to complete killing in the mucin group.

The next step in the Sinofresh Healthcare, Inc.’s pursuit of scientific data was to look at viral pathogens at the Lovelace Respiratory Research Institute in Albuquerque, New Mexico. The theory being tested was that the viral cell wall (or more correctly termed the capsid) had a similar chemical make up to the bacterial cell wall. This theory also extended to those strains of viruses classified as “enveloped” which have an additional layer surrounding the capsid. The company chose a good cross section of strains representing RNA viruses, DNA viruses, enveloped viruses and non-enveloped viruses. These studies were conducted first in-vitro and then again in vivo (mice). The results were similar to the MRSA studies in that the product exhibited an ability to kill the viral organisms by disrupting the capsid.

The most recent studies were conducted at Virion Systems in Rockville, Maryland. These studies were designed to evaluate the product’s efficacy in killing MRSA in living noses (animals) as well as to look at the effect on the “normal flora”. The results of this study were the most encouraging to date. Not only did the product virtually eliminate the MRSA but the normal flora, while slightly reduced, were left at a healthy concentration. These findings could be significant as institutions look for ways to effectively curb the spread of MRSA.